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RAP1 Is Essential for Silencing Telomeric Variant Surface Glycoprotein Genes in Trypanosoma brucei

  • Xiaofeng Yang
  • , Luisa M. Figueiredo
  • , Amin Espinal
  • , Eiji Okubo
  • , Bibo Li
  • Cleveland State University
  • Xi'an Jiaotong University College of Medicine
  • The Rockefeller University

Research output: Contribution to journalArticlepeer-review

136 Scopus citations

Abstract

Trypanosoma brucei expresses variant surface glycoprotein (VSG) genes in a strictly monoallelic fashion in its mammalian hosts, but it is unclear how this important virulence mechanism is enforced. Telomere position effect, an epigenetic phenomenon, has been proposed to play a critical role in VSG regulation, yet no telomeric protein has been identified whose disruption led to VSG derepression. We now identify tbRAP1 as an intrinsic component of the T. brucei telomere complex and a major regulator for silencing VSG expression sites (ESs). Knockdown of tbRAP1 led to derepression of all VSGs in silent ESs, but not VSGs located elsewhere, and resulted in stronger derepression of genes located within 10 kb from telomeres than genes located further upstream. This graduated silencing pattern suggests that telomere integrity plays a key role in tbRAP1-dependent silencing and VSG regulation. © 2009 Elsevier Inc. All rights reserved.
Original languageEnglish
Pages (from-to)99-109
Number of pages11
JournalCell
Volume137
Issue number1
DOIs
StatePublished - Apr 3 2009

Keywords

  • CELLBIO
  • DNA
  • HUMDISEASE

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